Treat Skin Allergies, Skin Rash, Itching, Infections, Fungus'Snigdhkanti' Neem - Sandal cream is a very effective way of treating skin infections. It is a completely natural and fine blend of neem oil, sandal oil and other herbal medicinal oils. Let us understand why it is so effective for skin problems: Neem Oil: The vital ingredient of this cream is Neem Oil. Neem oil obtained from crushing the seeds of the neem tree has a wide range of benefits. Neem preparations can get rid of Fungal Infections. Neem has been found to be deadly to 1. Candida), athlete's foot and . Neem is reported to be effective in treating certain Fungi that. Athlete's Foot fungus that infects hair, skin. Sandal Oil. This is the other important ingredient that has been used in the preparation of. Neem Cream. Sandalwood oil is extracted from the sandalwood tree. The American Journal of Gastroenterology (2007) 102, S271–S426; doi:10.1111/j.1572-0241.2007.01491. CLINICAL VIGNETTES. Walgreens.com - America's online pharmacy serving your needs for prescriptions, health & wellness products, health information and photo services. Offers vitamins and minerals, herbal supplements, health and beauty items, and weight loss products. Provides nutrition recommendations, resources, and company profile. The sandal. tree grows mainly in south India, and is used not only for medicinal purposes, but. Sandal for the Skin: Sandalwood is good for all skin types, in particular dry and oily skin and acne. Colon Cleansing Springfield Mo Real EstateShop for health care products at Walmart.com and browse vitamins, medicine cabinets and home health care products. Live better. The all natural neem -sandal cream provides an effective treatment for various skin conditions like allergies, skin rash, itching, infections, fungus and athlete. Hip Pain Info is an educational website designed to help people obtain information about hip anatomy and injuries. It is also. recommended for mature and tired skin as well as stretch marks and scars. Sandalwood paste is found to be effective as an effective remedy for prickly heat. Coconut and Olive oils soften the affected. Beeswax holds all these ingredients homogeneously keeping their natural properties. In all, the herbal and medicinal oils present in this cream have a powerful healing. Apply as required or 2- 3 times daily. If skin tends to become too oily, a paste. Herbomineral powder may. In all, the herbal and medicinal oils present in this cream have a powerful healing. The. steroid creams didn't let the rash appear (only if I stopped them for 3 days) but. Vaseline could. help not showing. Even after 8 hours of the first application. I used the cream 5 minutes ago, the nose. My grand daughter. I think it was a fungus. She has been on. prescription medicine for this problem but it did not help. The Neem- Sandal Cream. I received the cream in a short amount of time. Thank you ! Worked well - took care of my excema on my ear. The cream the Dr prescribed did not even. I received herbomineral and. I have been pleasantly. I will definitely pass the word along about how well this product. I look forward to ordering more from you in the future. It has already cured. It works within the day. The neem and sandalwood. Highly recommended! The product came safely from India. The Neem salve is really. I believe, prevent. Thanks for good products and good service! American Journal of Gastroenterology - CLINICAL VIGNETTESThe American Journal of Gastroenterology (2. S2. 71–S4. 26; doi: 1. Yarze, MD, FACP, FACG FASGE., *GI Division, GI Associates of Northern New York, Glens Falls, NY. Purpose: To describe a rare symptomatic complication of peroral colon lavage preparation. Methods: Colonoscopy is now frequently performed as a primary colorectal cancer screening modality. I recently cared for a patient who sustained a rare complication . Upon pre- procedure questioning, the patient mentioned that she had difficulty ingesting the colon lavage preparation, which included magnesium citrate, bisacodyl, and polyethylene glycol solution. Non- bloody vomiting occurred 1. The patient volunteered that the rectal effluent turned from yellow to blackish in color, during the night before which the procedure was performed. She was taking an NSAID intermittently, but this practice was discontinued 5 days prior to presenting for the procedure. There was no recent iron or bismuth ingestion. Physical examination showed slightly pale conjunctivae, but was otherwise unremarkable. We discussed also performing upper GI endoscopy, should it be deemed appropriate upon completing the colonoscopy. The patient was agreeable, and informed consent was obtained for both procedures. The colonoscopy was performed under moderate sedation. The examination was completed to the cecum and the mucosa was coated with melenic residue (which was suctioned and tested overtly positive for blood). The terminal ileal mucosa was similarly coated with black, bloody residue. The colonoscopy was otherwise normal (within the limits of the suboptimal preparation). Given these findings, upper GI endoscopy was then performed. At upper GI endoscopy, a prominent 3 cm nonbleeding Mallory- Weiss tear was found at the EG junction. No endoscopic therapy was deemed necessary, and she was discharged home on a proton- pump inhibitor (4 week course), with instructions to avoid ASA/NSAID use for 4 weeks. A CBC revealed a Hgb of 1. The patient recovered uneventfully. Conclusion: This case highlights a potential, albeit rare complication, which can be seen in association with peroral colon lavage preparation, which was administered prior to screening colonoscopy. Given that screening colonoscopy is now frequently performed, and nausea and vomiting are not a rare occurrence during the preparation, it is possible that such symptomatic events may occur with increasing frequency in the future. Top of page. 42. 7 . Yarze, MD, FACP, FACG FASGE., *GI Division, Gastroenterology Associates of Northern New York, Glens Falls, NY. Purpose: To describe an uncommon appearance of heterotopic gastric mucosa (HGM) in the cervical esophagus. Methods: An . HGM may be found in 1. Results: Over the years, I have found many patients who have had cervical esophageal HGM, which endoscopically appeared as 2 . One such case is shown in Figure 1. I have found that in patients with cervical esophageal HGM, the solitary island of gastric tissue is most commonly noted at the 7 to 8 o'clock position (which corresponds to the posterior position). In those in whom I've found . I would anecdotally estimate cervical esophageal HGM to have this less common appearance in 5% of cases in whom cervical esophageal HGM is detected. Conclusion: Cervical esophageal HGM can uncommonly present endoscopically as 2 . Yarze, MD, FACP, FACG FASGE, David M. Markowitz, MD., *GI Division, Gastroenterology Associates of Northern New York, Glens Falls, NY and Department of Radiology, Glens Falls Hospital, Glens Falls, NY. Purpose: To describe an uncommon presentation of hepatic hydrothorax. Methods: A 7. 7 yo male presented with recurrent massive bilateral pleural effusions. Background medical history included systemic hypertension, atrial fibrillation and a remote history of colon carcinoma. He was initially evaluated by my associate 9 months earlier, when abnormal liver biochemical tests were noted. At that time, he had hyperalkalinephosphatasemia (5 ULN). The serum transaminases were . The serum total bilirubin was normal and the albumin was mildly depressed at 3. Full serologic/virologic hepatic investigation was normal aside from the alpha- 1- antitrypsin phenotype revealing MS Pi- type heterozygosity. AMA was specifically negative. An ERCP was performed, which revealed . Liver biopsy revealed . He had no ascites at the time and was to be followed expectantly. He then presented with massive, symptomatic bilateral pleural effusions. He underwent multiple thoracenteses, which showed . Pleuroscopy showed no abnormality, and echocardiography was normal. Consultation was then requested after pleurodesis failed to alleviate the problem. Results: During this evaluation, abdominal CT revealed a . Ultrasound- guided diagnostic paracentesis of a 2 cm RLQ fluid collection confirmed a portal- hypertensive fluid (SAAG = 1. Doppler interrogation of the portal and hepatic venous systems revealed no thrombosis. Radionuclide scanning was performed after 6. Ci of TC9. 9m macroaggregated albumin was injected into the peritoneal cavity (right lower quadrant). This confirmed prompt tracer localization (at 4. Vigorous sodium restriction and combination diuretic therapy were unsuccessful in controlling the pleural fluid accumulation. The patient underwent TIPS placement, which alleviated his bilateral hepatic hydrothoraces. Conclusion: This case highlights an uncommon presentation of hepatic hydrothorax, where pleural fluid accumulation was both bilateral and occurred in the absence of significant ascites. Radionuclide instillation into the peritoneal cavity was employed to facilitate the diagnosis, and TIPS placement successfully alleviated the symptomatic pleural effusions. Top of page. 42. 9 . Yarze, MD, FACP, FACG FASGE., *GI Division, Gastroenterology Associates of Northern New York, Glens Falls, NY. Purpose: To describe the use of . Background history included diverticulosis and she ingested no ASA/NSAID or anticoagulants. Upon presentation, Hb was 1. The patient desired no evaluation, and immediately prior to planned hospital discharge, large volume, bright- red colored, painless hematochezia recurred. On this occasion, she became transiently hypotensive and the Hb fell to 6 g%. A bleeding scan suggested active bleeding from the left colon. EGD was normal. At colonoscopy, pancolonic diverticulosis was noted, but no active bleeding was seen, and ileal intubation was normal. A large adherent clot was seen in the mid- sigmoid. The clot was flushed away and a small- neck diverticulum was noted beneath. A tiny erosion was noted at the neck of the diverticulum, but no visible vessel could be seen. Epinephrine solution was injected at the neck of the diverticulum and the site was marked with ink, as it was thought to be a potential source of bleeding. Bleeding recurred 4. Repeat colonscopy again revealed no active bleeding and no clot was seen in the sigmoid region. A metal clip was placed in the region of the previously placed ink mark, for possible radiologic identification of this area of presumed hemorrhage. Hemodynamically significant bleeding recurred another 3. Results: At angiography, no bleeding site was initially seen on either the SMA or IMA . Hemorrhage was arrested with a third- order . Bleeding has not recurred after a 1. Conclusion: In cases of recurrent, . The former can be used to facilitate identification during repeat endoscopy or surgery, and the latter can be beneficial during angiographic investigation. Due to their expense, these maneuvers should not be employed routinely, but rather as an adjunct under special circumstances (e. Yarze, MD, FACP, FACG FASGE., *GI Division, Gastroenterology Associates of Northern New York, Glens Falls, NY. Purpose: To describe a novel approach utilized in a case of refractory, coexistent Crohn's disease and idiopathic thrombocytopenic purpura (ITP). Methods: Case Report: 5. Crohn's disease spanned 2 decades and she had previously undergone a partial colon resection. She was hospitalized on two recent occasions and was cared for by another gastroenterologist. Recent colonoscopy (done when the platelet count was 2. Crohn's colitis. She was glucocorticoid- dependent (5. She had reported questionable allergies/intolerances to all 5- aminosalicylate preparations, 6. MP, methotrexate, and metronidazole. Diffuse urticaria occurred after (4) infliximab infusions (despite an excellent clinical response, and administration of a . She was seen at a tertiary referral center for opinions from the IBD and hematologic perspectives. No novel approach was recommended, and a surgical team considered treatment of her IBD contraindicated (platelet count now 4,0. I was asked to see the patient after transfer back to our community hospital. She was having 1. Results: The platelet count remained 4,0. After discussion, I recommended a trial of oral 5- aminosalicylate, and budesonide was started, in anticipation of tapering of prednisone. Given prior response to infliximab, (but severe allergic reaction), a humanized anti- TNF monoclonal antibody was started (adalimumab 1. All the above interventions were tolerated and diarrhea improved. Six weeks later, she was discharged, having 5 soft, non- bloody bowel actions per day. Twelve weeks after starting adalimumab, prednisone was withdrawn. From the IBD perspective, she remains in clinical remission after 4 months. Conclusion: Transition to adalimumab was successful in achieving and maintaining clinical remission with simultaneous, treatment- refractory Crohn's disease and ITP, with previously demonstrated severe allergy to infliximab. The ITP remained untreatable, and this limited the ability to utilize more traditional medical (6. MP, MTX) and surgical therapies for the underlying IBD. Adalimumab . Yarze, MD, FACP, FACG FASGE., *GI Division, Gastroenterology Associates of Northern New York, Glens Falls, NY. Purpose: To describe a case of . Evaluation revealed mild hyperaminotransferasemia and low- titer (1. IU/ml), type 1b viremia.
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